By B. Pomeranz (auth.), Gabriel Stux M.D., Richard Hammerschlag Ph. D. (eds.)
There is a big bring up in curiosity in acupuncture via well-being care shoppers. the 3 major purposes for this are scientific effectiveness, price effectiveness and the credibility that medical trials and physiological learn have supplied. Acupuncture is swiftly relocating out of the sector of "alternative" drugs, largely back since it is grounded extra firmly than different replacement remedies in examine. The e-book offers the reader with the updated info at the scientific bases of acupuncture.
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Extra info for Clinical Acupuncture: Scientific Basis
1 and 2 . C. Takeshige 32 Dorsal hippocamcus Lateral hypothalamus I I Descending pain inhibitory system Pain inhibition Acupuncture point Fig. 2. Acupuncture analgesia-producing system Fig. 3a, b. Location of lesioned and stimulated sites in the arcuate nucleus. The arcuate nucleus is bounded by the broken line. The enlarged figure shows the sites at which lesions abolished AA. Analgesia produced by stimulation (SPA) at the open circles was abolished by hypophysectomy. Afferent SPA at the filled circles was not affected by hypophysectomy.
3 Synaptic Connections Between Acupuncture Afferent and Efferent Pathways [25, 28] The final region of the acupuncture afferent pathway is found in the M-HARN, which is anatomically close to the P-HARN, the initial region of the acupuncture efferent pathway (Fig. 3). Microinjection of the dopamine antagonist haloperidol antagonizes AA dose-dependently, while microinjection of dopamine into the P-HARN induces dose-dependent analgesia. , as the neurotransmitter at the interface between the acupuncture afferent and efferent pathways.
14). Beta-endorphin and ACTH released concomitantly from the pituitary gland might be the presynaptically acting neurohumoral factors in AA and non-AA, producing dopaminergic transmission in the P-HARN (Fig. 5). The possibility that ACTH acts as a neurohumoral factor in other regions of the non-AA afferent pathway has been examined by recording potentials evoked by nonacupoint stimulation after lesioning in the A-HARN, the final station of the non-AA afferent pathway. Such evoked-potentials in the A-HARN are enhanced by intraperitoneal administration of ACTH and are markedly decreased by hypophysectomy.