By Mark Findlay, Christopher Isles
This academic booklet teaches the reader on how top to debate and deal with acute and protracted displays of renal ailments. Nephrology research advisor: Tubules to Transplants is a useful source for junior medical professionals, scientific scholars and renal nurses who come across renal sufferers of their day-by-day perform. taking into account concise examining on particular themes this booklet acts as either a brief reference textual content and examine consultant. The format has been designed in a query & resolution structure that allows you to advertise self-directed learning.
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Additional resources for Clinical Companion in Nephrology
Org/guidelines/ modules/acute-kidney-injury. Rhabdomyolysis Q1 What is rhabdomyolysis and how does it present? The term rhabdomyolysis describes skeletal muscle breakdown regardless of the underlying cause. Clinical presentations range from an asymptomatic incidental finding on blood or urine testing, to muscle aches with dark coloured urine (myoglobin) through to a severe illness involving AKI requiring renal replacement therapy. The frequency of AKI in rhabdomyolysis is variable, being reported at 15–50 %.
The rest is stored intracellularly in tissues. It is therefore difficult to assess body stores from serum levels. Usual dietary phosphate intake is 30–50 mmol/day, mainly in dairy products. 4 mmol/l. Q2 Give the causes of hypophosphataemia These can be considered under three headings, summarised in the box below. common presenting features. 3 mmol/l is a medical emergency with risk of seizures, focal neurological deficits and heart failure, respiratory failure (due to weakness of the diaphragm), a proximal myopathy, dysphagia and ileus in addition to above.
Prostatic disease may be benign or malignant. Benign prostatic hypertrophy usually presents with nocturia, difficulty starting, poor stream and dribbling towards the end of micturition, and tends to cause bladder neck obstruction (bladder is full on ultrasound) while prostatic cancer more commonly invades the base of the bladder leading to obstruction at both ureters (bladder empty on ultrasound). Other important clues to obstructive uropathy are a history of renal stones. Bear in mind stone disease must be bilateral or present in a patient with a solitary kidney in order to cause AKI.