By Jerald Marifke M.D., Jay Sandlow M.D. (auth.), Parviz K. Kavoussi, Raymond A. Costabile, Andrea Salonia (eds.)
Clinical Urologic Endocrinology: rules for Men’s Health offers an geared up, available reference on men’s endocrinological future health.
Over 30 million males within the US on my own be afflicted by erectile disorder and over thirteen million males within the US be afflicted by hypogonadism (low testosterone). One out of 7 additionally be afflicted by subfertility of which 50-60% have male issue involvement. progressively more males are coming ahead to hunt remedy for such matters, which long ago have been thought of taboo and there's a powerful want for a e-book which gives counsel for practitioners who aid males of their reproductive and sexual matters. This ebook covers extensive the main concerns in male reproductive overall healthiness in a single easy-to-use source.
Clinical Urologic Endocrinology: ideas for Men’s Health is a helpful reference for urologists, endocrinologists, inner drugs physicians, relatives drugs physicians, intercourse therapists, and allied healthiness pros supplying deal with males within the parts of sexual overall healthiness, fertility, and men’s endocrinological future health.
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Additional resources for Clinical Urologic Endocrinology: Principles for Men’s Health
1997;16(3):303–6. 15. Layman LC, et al. Mutations in gonadotropin-releasing hormone receptor gene cause hypogonadotropic hypogonadism. Nat Genet. 1998;18(1):14–5. 16. Bhasin S. Approach to the infertile man. J Clin Endocrinol Metab. 2007;92(6):1995–2004. 17. Weiss J, et al.
Adult mullerian duct or utricle cyst: clinical significance and therapeutic management of 65 cases. J Urol. 2002;167:1740–4. 11. Dork T, Dworniczak B, Aulehla-Scholz C, Wieczorek D, Bohm I, Mayerova A, et al. Distinct spectrum of CFTR gene mutations in congenital absence of vas deferens. Hum Genet. 1997;100:365–77. 12. Brioude F, Bouligand J, Trabado S, Francou B, Salenave S, Kamenicky P, et al. Non-syndromic congenital hypogonadotropic hypogonadism: clinical presentation and genotype-phenotype relationships.
During the inguinoscrotal phase of testis migration, intra-abdominal pressure is thought to provide the main driving force for descent. However, evidence in rodents suggests the genitofemoral nerve (GFN) produces calcitonin gene-related peptide (CGRP), which stimulates contractions in the muscle fibers in and around the gubernaculum, further propagating this stage of testicular descent . During the inguinoscrotal phase, the processus vaginalis extends caudally as an outpouching of the parietal peritoneum, increasing in length as the testis descends to its final location.