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By Carole R. Mendelson

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Additional resources for Endocrinology of the Lung: Development and Surfactant Synthesis (Contemporary Endocrinology)

Sample text

Based on the properties of glucocorticoid stimulation, it is expected that the SP-B gene is a direct target for glucocorticoids and thus contains one or more GREs. The sequence data obtained by PilotMatias et al. 4 Transcription rates were determined by nuclear run-on assay after exposure of human fetal lung explants to cortisol (1 µM) for 28 h or for 24 h plus 4 h after media changes to remove the steroid. mRNA half-lives were calculated from time-course experiments using actinomycin D. Data from refs.

Premature delivery of foetal lambs infused with glucocorticoids. J Endocrinol 1969;45:515–523. 2. Seidner S, Pettenazzo A, Ikegami M, Jobe A. Corticosteroid potentiation of surfactant dose response in preterm rabbits. J Appl Physiol 1988;64:2366–2371. 3. Platzker AC, Kitterman JA, Mescher EJ, Clements JA, Tooley WH. Surfactant in the lung and tracheal fluid of the fetal lamb and acceleration of its appearance by dexamethasone. Pediatrics 1975;56:554–561. 4. Johnson JW, Mitzner W, London WT, Palmer AE, Scott R, Kearney K.

Am J Respir Cell Mol Biol 1991;4:449–454. 54. Xu J, Possmayer F. Exposure of rabbit fetal lung to glucocorticoids in vitro does not enhance transcription of the gene encoding pulmonary surfactant-associated protein-B (SP-B). Biochim Biophys Acta 1993;1169:146–155. 55. O’Reilly MA, Gazdar AF, Clark JC, Pilot-Matias TJ, Wert SE, Hull WM, Whitsett JA. Glucocorticoids regulate surfactant protein synthesis in a pulmonary adenocarcinoma cell line. Am J Physiol 1989;257:L385–L392. 56. O’Reilly MA, Clark JC, Whitsett JA.

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