By Andrea R. Genazzani, Basil C. Tarlatzis
This quantity represents an up to date assessment on ovarian capabilities and replica, supplying the reader with the most recent advances in gynecological endocrinology. It is released in the overseas Society of Gynecological Endocrinology (ISGE) sequence, and is predicated at the 2015 foreign university of Gynecological and Reproductive Endocrinology wintry weather direction. The ebook covers a truly wide variety of topics with specific concentration on ovulation and assisted replica, ovarian getting older and fertility, and untimely ovarian failure. The volume will be an invaluable tool for gynecologists, endocrinologists, obstetrician, and specialists in women’s wellbeing and fitness.
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Additional info for Frontiers in Gynecological Endocrinology: Volume 3: Ovarian Function and Reproduction - From Needs to Possibilities
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Fertil Steril 99:2000–2008 11. Borgbo T, Povlsen BB, Andersen CY, Borup R, Humaidan P, Grøndahl ML (2013) Comparison of gene expression profiles in granulosa and cumulus cells after ovulation induction with either human chorionic gonadotropin or a gonadotropin-releasing hormone agonist trigger. Fertil Steril 100:994–1001 12. Haouzi D, Assou S, Monzo C, Vincens C, Dechaud H, Hamamah S (2012) Altered gene expression profile in cumulus cells of mature MII oocytes from patients with polycystic ovary syndrome.
These spread to the granulosa cells (GC) where they are transformed to estrogens, by aromatisation, stimulated by the FSH. There is proof of the role of the As as positive regulators of follicular development with synergistic effects with FSH in folliculogenesis. The androgens regulate the function of the GC of the small antral follicles by upregulating the expression of FSH receptors (FSH-R). Treatment with testosterone (T) or dihydrotestosterone (DHT) increases FSH-R expression in the GC in monkey and promises the start of the primordial follicle growth.
C. Tarlatzis and C. Vaitsopoulou resistance to apoptosis and participates in endometrial stromal cell fate decision toward the end of the menstrual cycle [20]. According to Tapia et al. [21], at the secretory phase in the human endometrium, the mRNA levels of the complement system molecules C4b-binding protein (C4BP) and adipsin (complement component factor D, CFD) increase. It is postulated that the complement system might provide the uterine cavity with immunity against bacterial infection. In this sense, C4BP may protect the embryo where an increased expression of an inhibitor of complement system activation could reduce the chance of a misdirected complement attack to the embryo [21].