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By Peter A M Weiss; Donald R Coustan

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Additional info for Gestational Diabetes: Origins, Complications, and Treatment

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Additionally, obese women with a normal GCT had a twofold increased risk for delivering an LGA infant. 4, model 2). , Mild Maternal Hyperglycemia Increases Risk of Fetal Overgrowth in Minority Pregnant Women: Camden Study, paper presented at the 68th American Diabetes Association Scientific Sessions, San Francisco, CA, 2008. a Data are mean (SE) and adjusted for age, parity, ethnicity, gestational age at delivery, and cigarette smoking. b Model 1 was adjusted for age, parity, ethnicity, cigarette smoking, and infant gender and with nonobese normal GCT as a reference.

2004. Plasma Adiponectin Concentrations in Early Pregnancy and Subsequent Risk of Gestational Diabetes Mellitus. J Clin Endocrinol Metab 89:2306–11. A. 2004. Adiponectin Is Reduced in Gestational Diabetes Mellitus in Normal Weight Women. Acta Obstet Gynecol Scand 83:341–47. H. 2005. Maternal Plasma Adiponectin Concentrations at 24 to 31 Weeks of Gestation: Negative Association with Gestational Diabetes Mellitus. Nutrition 21:1095–99. 41. P. 2012. Hypoadiponectinemia during Pregnancy: Association with Risk of Varying Degrees of Gestational Hyperglycemia and with Maternal Ethnicity.

2, recent data support the influence of maternal obesity, pregnancy weight gain, increased oxidative stress, and elevated lipids (particularly free fatty acids) on insulin resistance during pregnancy, including GDM, and on foetal growth. 2 Summary of the link between maternal metabolism (in particular glucose levels and insulin sensitivity) and foetal growth. also are derived from the placenta and, together with the placental hormones, give rise to increased insulin resistance and act as factors to promote foetal overgrowth and adiposity.

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